The following article was written by Kreas
First of all there are three different types of gyno: Estrogen induced, Progesterone induced and Prolactin induced. Of course you can avoid all three types of gyno by keeping Estrogen within the normal range. The precursor to any type of gyno is Estrogen! Once you let Estrogen build up you signal to your brain that you have conceived, it doesn’t matter if you are a man or woman, your body at this point will have to go through certain processes to prepare you for lactation. Firstly your body will rush to use that Estrogen and build up breast tissue (which will form a lump) which is mandatory for the lactation process. Once this stage has been completed and you have let Estrogen still high your Progesterone will increase (Estrogen can still remain high) which is an attempt from your body to make the tissue larger and also make your aerolas bigger (puffy and enlarged nipples) again to get them ready for lactation. Last stage of gyno is Prolactin lactation, all previous stages were preparing the body for this moment at this point your Progesterone and Estrogen will drop and your Prolactin will spike, this is when someone starts lactating.
The most challenging hormone for the steroid user is Estrogen by far. It is the cause of any changes in your gyno/pecs, mood, libido, hardness, bloat, skin, prostate, appetite – you name it, when you feel off 90% of the time is due to low/high Estrogen levels.
When you hit your sweet spot you will know, you can’t miss it. You will feel happy, content, you will fuck like a champ, eat like a champ and train like a champ and too top it off everybody around you will be happy as well.
Here is an indicator I used since my early days on hormones.
Low Estrogen sides:
- Great erections
- Difficulty to ejaculate
- Dull orgasm
- High E2:
- Inability to maintain erection
- Great difficulty ejaculating
- Centered E2
- Easy erections
- Able to control ejaculation
- Stunning orgasm
Low and high Estrogen sides are very alike, the more experienced you get the easier it is to differentiate between them but it will always be tricky. If in doubt, get your Estradiol checked though blood work.
High Estrogen sides:
- Loss of libido
- Water retention (Bloat)
- Pissing less than the water you consume
- Moon face
- Very small testicles
- Scrotum hanging too high
- Soft testicles
- Extreme oiliness all over
- Soft erections
- Extreme cravings for sugar
- High blood pressure
- Blood pressure spikes
- Enlarged prostate
- Pressure in lower abdomen when urinating
- Thin stream of urine
- Low estrogen sides:
- Dry skin
- Dry lips
- Loss of libido
- Good morning wood
- Loss of wood while having sex
- Loss of sensitivity
- Dry gland (penis)
- White gland
- Loss of girth
- Mood swings
- Crying for no reason
- DHT rage (aggression you take out on others)
- Dull orgasm
- Hesitation just before urinating
- Night sweats
- Loss of appetite
- Constant fatigue
- Diuretic effect (pissing more water than you are consuming)
- Itchy scalp
- Obsessive thoughts
I got each and everyone of those sides over the past few years while I have been juicing and I am sure I am forgetting some potential sides. When you get one side effect, it is just an indication use this list to make a full picture. So say you have loss of libido, no zits and dry skin/flaking you know your E2 is low. Say you have loss of libido, acne, uncontrollable aggression, and bloat you know your E2 is too high. Never go by one side only, being bloated only means nothing, having dry skin only means nothing again.
Keep in mind Estrogen is good for you in many ways (libido, mood, skin quality, hair, nails etc). But, most importantly, Estrogen is good for your liver. I am sure you have heard how Arimidex and Letrozole are bad for your liver values while Aromasin is ‘better’, in reality all AI’s are as bad as each other for your liver values. The more you lower Estrogen, the worse your liver values will get – it doesn’t matter which AI you use, all that matters is how much you are lowering your Estrogen. If you lower your Estrogen by 10nl/dl you won’t notice much difference. If you crash your Estrogen down to single digits I guarantee you that your HDL/LDLl will be completely out of whack no matter which AI you used.
Suicidal AI vs Non-Suicidal/Binding AI
Arimidex and Letro are non-suicidal AI’s, all they do is bind any Estrogen you convert directly on your aromataze enzyme. Each AI binds a different percentage of Estrogen, Letro binds more than Arimidex of course. The problem with binding AI’s is that once you cease use, all of the Estrogen that had accumulated over the weeks/months you were using that AI suddenly gets released – this process is called Estrogen rebound and I am sure you know it can be far worse than Estrogen while on a cycle since normaly when you drop your AI you either cruise with a low dose of Test or PCT. In both cases you have far less Test in you and once all that Estrogen is released you got a much higher chance of getting gyno and of course you are going to be bloated like a balloon and feel soft for weeks till your Estrogen comes down to normal levels.
Aromasin is the new generation of AI and it is suicidal, the difference between Aromasin and the other AI’s is Aromasin will actually destroy/kill a certain percentage of your aromataze enzyme so by doing so it also kills any estrogen that was attached to that enzyme. This means when you stop using Aromasin you won’t rebound at all like you would with the binding AI’s. If anything you will have to wait for a while for your body to start producing more aromataze (very bad if you crashed your Estrogen comparing to using the other AI’s). Each person is different in the rate they create new aromataze, for me it takes around 2 weeks, for someone else it can take one or three weeks. The only way you can speed up the process is by using HGH, you can use all the Dianabol you want, all the Test Suspension you want but if you crashed your Estrogen with Aromasin and you don’t have aromataze you wont even bloat from those compounds – there will not be any Estrogen conversion, you won’t even get any results from the Dianabol.
Arimidex will lower your Estrogen by about 50-60%. Of course, if you keep taking it that percentage accumulates so you lower 50% by another 50% and so on, you can easily end up with your Estradiol in the singles if you take it for long enough at a high enough dose and you aren’t converting much Estrogen from aromatizing gear (using low dose of Test and high dose AI). Arimidex, in my opinion, is the best suited for TRT purposes, this is because the rate in which it lowers Estrogen compared to the other AI’s is smaller. For TRT purposes you only need 1mg of total Arimidex per week to keep Estrogen in range. E2 = 20-25ng/dl = sweet spot.
Why Arimidex is bad for blasting
Arimidex is not particularly effective for drugs that tend to be subject to peripheral aromatization, Methandrostanolone in particular. Often a full 2mg daily of Adex will still not stop Dianabol bloat and cramping. Arimidex is good at E1 suppression (tissue affinity, gonadal, adrenal, etc and because its a competitive inhibitor). It suppress E1 at even the lowest of doses, but takes rather high doses to see significant impact on peripheral aromatase.
While cutting with a low Test dose and non-aromatizing gear it is as good as an AI as any but while bulking with compounds that aromatize heavily Arimidex is the worst AI you can use. No matter how much you use you will still be much more bloated than you would by using Aromasin/Letrozole.
Common dose while blasting: 0.5mg ed/eod
TRT dosage: 0.25mg eod, 1mg e6-7d
Aromasin is hands down the best down AI for blasting but it does have its downsides. I found that the more you use Aromasin, the more senstitive you become to it. When starting out with Aromasin even 25mg per day is a common dose for a mild cycle of say 750mg Test and 500mg Deca. As time goes by, the more you use it the less Aromasin you will actually need. You will end up needing 12.5mg EOD (if not less) for a mild cycle. It doesn’t happen in a few days, it takes months. Another drawback of Aromasin is hairloss, compared to the other AI’s I found it makes me shed a lot more. One side effect of aromasin is Alopecia. The other two AI’s have hairloss/hair thinning as a side effect but not full blown Alopecia.
Like I mentioned earlier, the biggest fear with Aromasin is crashing Estrogen too low. At this point all you can do is wait or up your HGH dose. Give it at least 10 days before you start taking any more AI even if you are switching to Arimidex.
The best write up about aromasin which reflects my experience with it 100% is here:
Aromasin – The king of anti-estrogens
This post is kind of long, but take the time to read it, it’s probably the most important thing you’ll ever read if you’re a bodybuilder.
Exemestane, sold under the name Aromasin by Pfizer, is an orally available suicidal aromatase inhibitor.
Because Exemestane is steroidal this gives it a favorable Estrogen suppression profile and confers a few really awesome benefits over other anti-estrogens both on paper and in real experience. Steroidal anti-estrogens have the benefit of being lipid-friendly and they all lower SHBG which increases the ratio of free to bound Testosterone, which as many experienced bodybuilders know can have a relatively profound, positive impact on gains.
I think it is important to understand how drugs work in order to properly dose them, Exemestane is a suicidal aromatase inhibitor, this means that it binds with aromatase enzymes and as it does so permanently disables the enzyme and destroys it. Hence the “suicidal” this chemical is like a kamikaze pilot out to destroy your aromatase enzymes which is what makes it so special.
Exemestane’s half life in the male body is actually very short (~9 hours) and it is quickly eliminated, however, since as soon as it enters your bloodstream it quickly destroys 80-90% of the aromatase enzymes present in your body, it is effective in maintaining significant reductions in estrogen for up to 72 hours after a single 25mg dose. Estrogen levels only begin to rise again after your body has begun to make new aromatase enzymes to replace the ones destro by exemestane.
There is a great study on the pharmacokinetics of exemestane in men which found the following:
- 24 hours after one 25mg dose estrogen levels are reduced by 70-80%
- 72 hours later estrogen levels are still 40% below baseline even though the drug itself is almost completely eliminated
- 120 hours after initial dose estrogen levels return to baseline (without rebounding)
This means that you can find the timing and dosage that works for you, I’ve seen some guys recommend between 25mg ED and 12.5mg e4d, and you can see why both are effective while providing different levels of estrogen suppression, and it is this flexibility that makes Exemestane such a versatile Anti-E.
BUT WAIT, there’s more. Aromasin is also a badass PCT drug! In males exemestane was found to increase total testosterone by ~60% after 10 days @ 25mg/day, however the same study found that while it increased total testosterone by 60% free testosterone was increased by over 100 percent! that’s right, it DOUBLES bio-available testosterone (natty of course).
I can tell you this much, when I take aromasin for PCT the results are dramatic, honestly my Libido is never absent at any point during PCT and I absolutely feel great within a matter of days, and this is taking 12.5mg ED, the only side effect i notice is stiff joints and other stiff areas
- Powerful aromatase inhibitor capable of stopping gynecomastia completely on its own (for aromatizing compounds)
- Has powerful bloat-reduction effects
- Lowers SHBG, increasing free test & makes all other anabolic steroids more bio-available (read: more gains)
- Can actually boost Libido on and off cycle
- Increases IGF-1
- No adverse changes in lipid profiles for men (granted if you are using it on cycle this may be different)
- Is NOT liver toxic
- No Estrogen rebound
- Typical aromatase inhibitor issues here include stiff joints and possibly lethargy
- More difficult to come by than Arimidex or Letro
Appropriate uses for Exemestane:
- On cycle Estrogen control – that’s right, any and all Estrogen related problems can and should be corrected with this compound, from gynecomastia to acne to bloat exemestane is a panacea, run it at 12.5mg e4d for gynecomastia protection and bloat control, or run it at 25mg ED for pre-contest or for gynecomastia sensitive individuals or moon face. the beauty of aromasin is it’s okay to use preventatively and not just as spot treatment for gynecomastia as it doesn’t hurt gains nearly to the degree that other Anti-E’s do, I’d still recommend using Anti-E’s only if you need them, but if you must use one throughout your cycle, you couldn’t pick a better compound to use.
- PCT – Aromasin is the premier PCT drug in my experience… honestly PCT is kind of fun with aromasin (maybe that’s a stretch) but it’s a breeze compared to clomid/nolva and significantly better than a-dex (more powerful and fewer sides) it works excellently with HCG – human chorionic gonadotropin – and keeps the extra aromatization from the HCG – human chorionic gonadotropin – injects at bay (you can even run higher dosages of HCG – human chorionic gonadotropin – above 500iu/inject) and another bonus is since it’s safe and comfortable to run for longer periods of time, you can stretch your PCT out to 6 or 8 weeks for suppressive cycles to make sure you get everything back in full working order
- Gynecomastia reversal – in conjunction with a selective estrogen receptor modulator (raloxifene or tamoxifen) and/or a dihydrotestosterone derived compound aromasin can be effective in reversing/reducing existing gynecomastia
- Off cycle Testosterone boost – sometimes if I don’t feel like running a cycle but still want a little extra kick I’ll take 25mg EOD for 4-6 weeks, gains aren’t improved all that greatly but significantly, but I do it more for the Libido/mental effects anyways.
- Hypogonadism – so you’re getting older, you’ve been cycling since you were 21 and your natural Test levels just never get back in the good range, but you don’t wanna go HRT? Aromasin will get you back in the game without having to take the plunge for HRT.
Common dosages: 12.5mg ED/EOD, 25mg ED/EOD
TRT dosages: 6.25mg ED, 12.5mg 2-3 times per week
This is an AI you can do without. It is by far the harshest of all AI’s, not necessarily because your Estrogen will be too low but because Letrozole as a compound/active ingredient is really harsh.
Ever climb up the stairs and felt as if you were dying same as a 500lb man would after taking two steps? That’s what Letro can do to you. My view on this is that it affects ones triglycerides, if you use letro long enough at max dose your triglycerides will be so high that even after climbing ten steps you will be struggling to breath.
Only application of Letro (which can be avoided/substituted with Aromasin) is for contest prep, I would never use it for either bulk, cut or gyno reversal as there are too many side effects for very little gain.
Also ever took Letro and still had nipple sentivity? Wonder why? Letro lowers SHGB dramatically, this allows free Testosterone to spike and as a result free Estrogen, this is the reason the Letro gyno reversal protocol doesn’t work (especially when its suggested to use it for one week only). In order to have low free Estrogen (Which AI’s cant lower) you need to drop your total Estrogen low. However everyone trying to reverse gyno already have high Estrogen and the moment you add Letro you have a ton of free Estrogen in your blood stream, which can make your gyno worse. To protect against free Estrogen you need a SERM, that’s why you cant have gyno reversal without a SERM since all AI’s lower SHGB.
Keep in mind you can’t use Nolvadex with Arimidex or Letro as you minimize their efficiency by 40%. That doesn’t work vice versa though, Nolvadex efficiency stays at 100%.
Common dosage: 0.62mg ED/EOD, 1.25mg ED/EOD
Keep in mind all 3 SERMs will work in favor of your liver (Agonists) since they are mild Estrogens, like I said earlier Estrogen is good for your liver so adding a SERM will always improve your HDL/LDL. All SERMs don’t lower Estrogen, in fact they will increase your total Estrogen. They also block your Estrogen in the nipple area, but similar to the binding AI’s once that Estrogen gets released you rebound and you end up with even higher Estrogen than before.
Agonist: Liver, uterus (female)
As I am sure you heard Nolvadex reduces IGF-1 levels by 25%. Now, that might seem like the biggest disadvantage ever but if you take in to account that your liver is going to be healthier while you use it, it balances out the deduction of your IGF-1 levels.
Nolvadex is more suited for PCT purposes rather than on cycle therapy (OCT) as it increases natural Test levels by 60% and decreases IGF-1 levels.
Dosage on cycle: 20-40mg per day
Agonist: Liver, bone (increases bone density like Deca and is a recognised treatment for osteoporosis)
Antagonist: Breast/nipple (stronger than Nolvadex for gyno)
Raloxifene doesn’t affect IGF-1 levels whatsoever, also it increases bone density but does nothing for your tendons like Deca does, so it might be a double edge sword if you are not using Deca along side it since by having stronger bones and muscles chance for tendon injury is higher.
Raloxifene is the ideal SERM for OCT since its an agonist for your bones, doesn’t affect IGF-1 levels and is perfectly safe to run with a 19 NOR. Raloxifene shouldn’t be used in PCT since it raises natural test levels by 40% only, 20% less than Nolvadex.
Dosage on cycle: 60mg-120mg per day
Nolva vs Raloxifene for HGH/IGF-1
Objective: The aim was to compare the impact of raloxifene and tamoxifen on GH-IGF-I and gonadal axes in healthy men.
Design: We conducted a randomized, open-label crossover study.
Patients and Intervention: Ten healthy men were randomized to 2-wk sequential treatment with tamoxifen (10 and 20 mg/d) and Raloxifene (60 and 120 mg/d), with a 2-wk intervening washout period.
Main Outcome Measures: We measured the GH response to arginine and circulating levels of IGF-I, LH, FSH, testosterone, and SHBG.
Results: Tamoxifen, but not Raloxifene, significantly reduced IGF-I levels by 25 ? 6% (P < 0.01) and increased SHBG levels by 20 ? 7% (P < 0.05) at the higher therapeutic dose. There was a nonstatistically significant trend toward a reduction in the GH response to arginine with both SERMs. Both drugs significantly increased LH, FSH, and testosterone concentrations. The mean increase in testosterone (40 vs. 25%; P < 0.05) and LH (70 vs. 30%; P < 0.01) was significantly greater with tamoxifen than with Raloxifene treatment.
Conclusions: Tamoxifen, but not Raloxifene, reduces IGF-I levels. Both SERMs stimulate the gonadal axis, with tamoxifen imparting a greater effect. We conclude that in therapeutic doses, Raloxifene perturbs the GH and gonadal axes to a lesser degree than Tamoxifen.
Nolva vs Raloxifene for gyno
Objectives: To assess the efficacy of the anti-estrogens tamoxifen and Raloxifene in the medical management of persistent pubertal gynecomastia. STUDY
Design: Retrospective chart review of 38 consecutive patients with persistent pubertal gynecomastia who presented to a pediatric endocrinology clinic. Patients received reassurance alone or a 3- to 9-month course of an estrogen receptor modifier (Tamoxifen or Raloxifene).
Results: Mean (SD) age of treated subjects was 14.6 (1.5) years with gynecomastia duration of 28.3 (16.4) months. Mean reduction in breast nodule diameter was 2.1 cm (95% CI 1.7, 2.7, P <.0001) after treatment with tamoxifen and 2.5 cm (95% CI 1.7, 3.3, P <.0001) with raloxifene. Some improvement was seen in 86% of patients receiving tamoxifen and in 91% receiving Raloxifene, but a greater proportion had a significant decrease (>50%) with Raloxifene (86%) than Tamoxifen (41%). No side effects were seen in any patients.
Conclusion: Inhibition of estrogen receptor action in the breast appears to be safe and effective in reducing persistent pubertal gynecomastia, with a better response to Raloxifene than to tamoxifen. Further study is required to determine that this is truly a treatment effect.
Clomid is a really harsh drug it should be avoided at all costs, if you get the visual sides/blurry vision from clomid they stay for life. They are rare but do happen.
Clomid should only be used in restart protocols under the supervision of a doctor. So if you want to start producing sperm again you will have to take HCG along with Clomid for 9-12 months straight. It has some use in PCT but it can be completely avoided by using SERMs only or ideally Nolvadex + Aromasin.
Some good info on clomid:
Clomiphene is a mixed agonist/antagonist. This is due to the fact that Clomiphene is composed of two isomers: enclomiphene (trans-clomiphene) and zuclomiphene (cis-clomiphene). Enclomiphene is an Estradiol receptor antagonist. Zuclomiphene is an estradiol receptor agonist. In all likelihood, the net antagonist effect might be due to the composition being 70% trans (enclomiphene) and 30% cis (zuclomiphene). Tamoxifen is more of a strict antiestrogen, decreases the effect of estrogen in the body, and potentiates the action of clomiphene. This combination came about after 100s of clinical experience.
So Tamoxifen is more of an antagonist, than Clomid is. Its better at blocking the ER than Clomid is. Clomid also seems to exert agonistic effects in parts of the brain that control emotion. That would explain why some turn into women on periods during there experiences with Clomid.
Tamoxifen is also made of slightly more isomers, the cis isomer of tamoxifen (inactive one) trans-tamoxifen and trans-4-OHT isomer.
- Clomid will double LH at 100mg/ED in 5-7 days and increase FSH by 20-50%. LH rises quickly post cycle, but not that quick.
- Clomid will raise enodgenous testosterone (total) by 146% after 3 months at 25mg/ED.
- Clomid at 100mg/ED will raise endogenous testosterone (total) by 268% after 8 weeks and free testosterone by 1,410% (That’s not a typo).
- Tamoxifen increased serum testosterone to 142% of baseline in only 10 days. It took 150mg/ED of Clomid to get the same 142% increase. After 6 weeks it raised testosterone and LH levels to an average of 183% and 172% of starting values.
Another thing to note after the above study is how sensitive the pituitary become to GnRH. The more sensitive the pituitary is to GnRH, the more LH it will produce. Tamoxifen increase pituitary sensitivity to GnRH and Clomid seemed to decrease it.
- Estrogen will decrease sensitivity to GnRH. It will not increase it. If estrogen were to increase the pituitary to GnRH it calleds “estrogen priming”. Priming the pituitary to become more sensitive to GnRH. This happens in females, but not males. There is no evidence to suggest there is E priming in males.
- Tamoxifen is more an antiestrogen than Clomid is. Both are SERM’s and selective with agonistic/antagonistic effects in “selective” tissues. Both will block the ER in breast tissue. Both are agonists in the liver, which would explain the increase in IGF binding proteins and decrease in plasma IGF.
Dose while on cycle: 50mg EOD. Some say it can substitute HCG but I haven’t tried that route.
Caber will lower both progesterone and will inhibit prolactin/lactation. It’s a dopamine agonist means it wont allow your body to lactate since it will occupy your dopamine receptors which are responsible for lactation. Caber is the perfect prolactin support when running any 19 NOR since the side effects are minimal – no drowsiness, doesn’t affect sleeping patterns and in general as far as dosing goes is far more flexible than Pramipexole or Bromo. Also there is no withdrawl when ceasing use of Caber like with Pramipexole
Caber is a regognised ED med, it reduces downtime (not to be confused with multiple orgasm) so if you need 24 hour recovery between sessions two weeks after taking Caber you will see a significant decrease in downtime you will need 12-16 hours to be ready for the next session, if you need 2 hours you will need 1 hour with Caber.
Also its known for the multiple orgasm effect, so when you ejaculate you will feel as if you are releasing two or three loads at the same time. This needs some input for the user though its not instant, the more you hold it in the more orgasms you will have in the end. Also without caber say you are having sex and you let some cum slip through you got a big chance to loose your hardon, with caber even if you let quite a bit slip through your hardon will become stronger and stronger and your climax will be insane. In this sense you can actually have 4-5 small orgasms coupled with a huge orgasm in the end that will feel as two more orgasms put together. Again it needs practice and self control from the user
Common dose on cycle: 0.25-0.50mg E3D
Common does to stop lactation: 1-1.5mg E3-5D
Prami like Caber will decrease progesterone and will inhibit prolactin/lactation. It’s a dopamine agonist like Caber so it will occupy dopamine receptors which are responsible for lactation.
Prami is a very peculiar drug! You need to taper up really slowly to get to the desired dose and also taper down really slowly to avoid the mild withdrawal effect it will cause. Prami is an addictive substance I wouldn’t recommend it for any cycle over 8 weeks the more you use it the harder it will be to come off it, also you will find you will want to increase the dose to maintain the ed effect. Prami’s ED effect is nowhere near as good as Caber. It does reduce downtime like Caber does but that’s about it there is not enhancement in your orgasm or your libido contrary to caber. Only advantage of Prami over caber is that if taken at the right time (2-3 hours) before bedtime it can work as good as a benzo to knock you out to sleep. Which when running Tren is a bonus. If however you dose it wrong (unwillingly of course) say 30 minutes – 1 hour before bed time you will find that after 2-3h of sleep you will be wide awake and probably sweating since the dopamine you suppressed 4 hours ago rebounds and you feel as if you just had a hit of coke in your sleep, not a good feeling. Also every time you up the dose it takes some adjusting even if you are used to the substance. I found that overtime I would up the dose for the first few nights I would sleep very light almost like sleeping awake that’s how it felt.
Sleep sides like vivid dreams and waking up mid night can be avoided by taking prami at the right time so you got to experiment with this (the earlier you take it the better). Make sure you never take prami in the morning or too early in the evening you are going to feel drained, dizzy, nauseous and like a zombie all you will think its when the time comes to go to sleep.
The worst part with prami starts when you quit, for the first few days after you quit, you will wake up in your sleep many times as if you were quitting cigarettes or weed even, then you will have the lightest sleep ever as if you were sleeping with your eyes open and the dreams will be negative and intense. Basically you get all the Prami sides you had earlier only they cant be avoided since you don’t take Prami anymore. This will subside completely after 5 – 7 days.
Common dose on cycle: taper up from 0.125mg to 0.25mg-0.50mg (the high dose only if you are stacking two 19nors or high dose of tren). After you are done with your cycle taper down even slower from 0.50mg to 0.125mg and stay one week on each increment then quit. No matter what you do expect some discomfort the first 3-5 days after you quit
Dose to stop lactation: You would probably need 1-2mg per day to stop lactation but I wouldn’t recommend it, it would take ages to rump up to that dose, if you are already lactating use caber worse thing that could happen when jumping to a high dose of caber would be to get a flush face that lasts 12-14h (annoying but much better than puking your guts of for hours).
I use firstname.lastname@example.org – they stock pharmaceutical grade AI’s. Shipping to the UK usually takes 1-2 days and they do ship internationally too. Just send them an email explaining that you got their email address from this website and ask for a price list and they will send over a full list of products and prices.